Drosophila melanogaster

Wolbachia has been reported both to extend and shorten longevity; its lifespan-modulating effects are proposed to involve pathways like insulin/insul…

Wolbachia infection affects differential gene expression in Drosophila testis, upregulating genes involved in carbohydrate metabolism, lysosomal degr…

Lactobacillus may be able to ameliorate the pH of the acidic midgut region by releasing weak bases, and also has a complex relationship with physiolo…

Acetobacter may be able to ameliorate the pH of the acidic midgut region by releasing weak bases, and also has a complex relationship with physiologi…

Wolbachia upregulates the expression of Pale and Ddc, mediates the expression of dopamine-related genes, increases total sleep time, and decreases sl…

The existence of Acetobacter has a balancing effect on food ingestion when carbohydrate levels are high in warmer months, stabilizing fitness compone…

A specific strain of Wolbachia was observed to reduce the initiation of aggressive encounters in Drosophila males, which may be achieved by influenci…

Wolbachia infection changes the expression of several genes associated with spermatogenesis, immunity (kenny), and metabolism (CG4988-RA) in the larv…

Listeria monocytogenes infection disrupts host energy metabolism, reduces antioxidant defense, and alters amino acid metabolism, accompanied by melan…

Wolbachia popcorn begins massive proliferation in the adult, causing widespread degeneration of tissues (brain, retina, muscle), culminating in early…

Spiroplasma poulsonii protects its host against parasitoid wasps and nematodes through the action of toxins from the Ribosome Inactivating Proteins f…

Wolbachia protects Drosophila melanogaster against two naturally occurring and virulent viral pathogens: La Jolla virus (LJV) and Newfield virus (NFV…

Wolbachia pipientis increases the recombination rate observed across two genomic intervals and increases the efficacy of natural selection in hosts.

Spiroplasma poulsonii supports the hypothesis that competition for host lipids underlies symbiont-mediated protection against parasitoid wasps.

Wolbachia enhances nanos expression via the bacterial TomO protein in order to fuel germ stem cell maintenance in infected Drosophila females.

Spiroplasma causes neurodegenerative phenotypes and a reduced life span in old flies compared to uninfected Drosophila melanogaster controls.

Spiroplasma poulsonii co-opts the yolk transport and uptake machinery to colonize the germ line and ensure efficient vertical transmission.

Wolbachia regulates the host's transcriptional response to viral infection and affects viral replication in Drosophila melanogaster.

Lactiplantibacillus plantarum could effectively inhibit fungal spore germinations on the body surfaces of Drosophila melanogaster.

Photorhabdus luminescens produces toxin complex (Tc) toxins as major virulence factors against the host Drosophila melanogaster.

Gut bacteria (Metagenome data) are necessary for the nutritional needs and survival of the parasite of Drosophila melanogaster.

Gut bacteria (Metagenome data) are necessary for the nutritional needs and survival of the parasite of Drosophila melanogaster.

Saccharomyces cerevisiae results in the development of larger ovaries and increased egg numbers in Drosophila melanogaster.

Wolbachia pipientis influences octopamine metabolism in Drosophila females, which is dependent on the symbiont genotype.

The virulent Wolbachia wMelPop infection improves the learning and memory capacity of the host, Drosophila melanogaster.

Acetobacter malorum results in the development of larger ovaries and increased egg numbers in Drosophila melanogaster.

Body-surface microbiotas (Amplicon data) can defend Drosophila melanogaster flies against fungal parasitic infections.

The presence of Spiroplasma (together with Wolbachia) in D. melanogaster up-regulated certain immune-related genes.

The presence of Wolbachia (together with Spiroplasma) in D. melanogaster up-regulated certain immune-related genes.

Lactobacillus plantarum FlyG1None.1.5 increases its growth-promotion ability by adapting to the Drosophila diet.

Lactobacillus plantarum FlyG1None.1.9 increases its growth-promotion ability by adapting to the Drosophila diet.

Lactobacillus plantarum FlyG2None.1.2 increases its growth-promotion ability by adapting to the Drosophila diet.

Lactobacillus plantarum FlyG2None.1.4 increases its growth-promotion ability by adapting to the Drosophila diet.

Lactobacillus plantarum FlyG2None.2.2 increases its growth-promotion ability by adapting to the Drosophila diet.

Lactobacillus plantarum FlyG2None.2.6 increases its growth-promotion ability by adapting to the Drosophila diet.

Lactobacillus plantarum FlyG11.1.2 increases its growth-promotion ability by adapting to the Drosophila diet.

Lactobacillus plantarum FlyG11.1.6 increases its growth-promotion ability by adapting to the Drosophila diet.

Lactobacillus plantarum FlyG11.2.6 increases its growth-promotion ability by adapting to the Drosophila diet.

Spiroplasma poulsonii manipulates the host's reproduction by killing its male progeny at the embryonic stage.

Lactobacillus plantarum FlyG2.1.8 increases its growth-promotion ability by adapting to the Drosophila diet.

Lactobacillus plantarum FlyG3.1.8 increases its growth-promotion ability by adapting to the Drosophila diet.

Lactobacillus plantarum FlyG7.1.6 increases its growth-promotion ability by adapting to the Drosophila diet.

Lactobacillus plantarum FlyG8.1.1 increases its growth-promotion ability by adapting to the Drosophila diet.

Lactobacillus plantarum FlyG8.1.2 increases its growth-promotion ability by adapting to the Drosophila diet.

Lactobacillus plantarum FlyG9.1.4 increases its growth-promotion ability by adapting to the Drosophila diet.

Lactobacillus plantarum FlyG9.2.5 increases its growth-promotion ability by adapting to the Drosophila diet.

Accharomyces cerevisiae (Fungi) mediates Drosophila melanogaster attraction, oviposition, and development.

Spiroplasma poulsonii MSRO-H99 expresses a protein, designated Spaid, which induces male killing.

Spiroplasma poulsonii MSRO-SE expresses a protein, designated Spaid, which induces male killing.

Erwinia carotovora carotovora 15 decreases olfactory discrimination in Drosophila melanogaster.

Wolbachia pipientis increases the heat stress resistance of the host Drosophila melanogaster.

Lactobacillus plantarum has the potential to reduce IMI-induced susceptibility to infection.

Gut bacteria increase the chemotaxis response to odorants in Drosophila melanogaster larva.

Gut bacteria reduce the memory of olfactory appetitive learning in Drosophila melanogaster.

Lactobacillus plantarum may be beneficial in reducing in vivo Chlorpyrifos (CP) toxicity.

Wolbachia strain wMel changes the composition of gut commensal bacteria in the fruit fly.

Spiroplasma poulsonii MSRO_BK is a male-killing Spiroplasma bacterium.

Spiroplasma poulsonii MSRO is a male-killing Spiroplasma bacterium.

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